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Dihydromyricetin (DHM) is a hepatoprotective flavonoid extracted from Hovenia Dulcis, a Japanese raisin tree that's famous in the traditional medicine world. It's been used since the year 659 throughout Japan, China, and Korea as a hangover cure.
Dihydromyricetin is an extract of grape vine tea, and is the main active component of flavonoids in rattan tea. Myricetin is a special kind of flavonoids, which can relieve alcohol poisoning, prevent alcoholic liver, fatty liver, inhibit the incidence of hepatocellular carcinoma, anti-hypertension, inhibit platelet aggregation in vitro and thrombosis in vivo, reduce blood lipids and blood sugar. It has special effects in improving the activity of SOD and protecting the liver.
1. Antibacterial effect: Pharmacological experiments show that dihydromyricetin has antibacterial effects on Bacillus subtilis, Staphylococcus aureus, Salmonella, Escherichia coli, Aerobacter, Saccharomyces cerevisiae, Rhodotorula mucus, Penicillium, Aspergillus niger, Aspergillus flavus , Mucor and Rhizopus all have bacteriostatic effects, especially on Gram-positive, Gram-negative cocci or bacilli.
2. Regulating effect on blood sugar and blood lipids: Pharmacological experiments in mice by gavage show that dihydromyricetin can significantly inhibit the increase of blood sugar in mice induced by alloxan, epinephrine, streptozotocin, etc., and at the same time increase serum insulin At the same time, the infiltration of lymphocytes in pancreatic tissue was significantly reduced, the inflammatory response was significantly reduced, and the number of pancreatic islets was significantly increased. Dihydromyricetin can reduce the content of serum triacylglycerol (TG) in streptozotocin-induced hyperglycemia rats, but has no significant effect on the content of serum total cholesterol (TC) and high density lipoprotein cholesterol (HDL2C).
3. Hepatoprotective effect: Dihydromyricetin has significant protective effect on rat hepatocytes from carbon tetrachloride poisoning injury, D2 galactosamine and lipopolysaccharide-induced liver injury in mice. Dihydromyricetin can protect the liver, accelerate the rapid decomposition of acetaldehyde, a metabolite of ethanol, and turn it into a non-toxic substance, reducing its damage to liver cells. In addition, dihydromyricetin can improve the increase of serum lactate dehydrogenase activity caused by liver cell injury, inhibit the formation of collagen fibers in hepatic M cells, so as to protect the liver and protect the liver, and greatly reduce the damage of ethanol to the liver. The normal state of the liver can be quickly restored. Microherb test proves that dihydromyricetin can protect the liver, accelerate the rapid decomposition of acetaldehyde, a metabolite of ethanol, into non-toxic substances, and reduce damage to liver cells. Dihydromyricetin works quickly and has a long-lasting effect.
4. Antioxidant effect: Dihydromyricetin with a purity of 98% can significantly inhibit the formation of malondialdehyde (MDA) in rat myocardial, liver and brain tissue homogenates, and inhibit MDA with the increase of dihydromyricetin concentration The effect of generating, 99% dihydromyricetin on the scavenging rate of diphenyltrinitrophenylhydrazine (DPPH) free radicals in the test system was increased. Dihydromyricetin can significantly inhibit the formation of MDA in oil, and the antioxidant effect is enhanced with the increase of dihydromyricetin purity (60% to 90%); it has strong antioxidant effect on animal and vegetable oils.
Dihydromyricetin (DHM) is a hepatoprotective flavonoid extracted from Hovenia Dulcis, a Japanese raisin tree that's famous in the traditional medicine world. It's been used since the year 659 throughout Japan, China, and Korea as a hangover cure.
Dihydromyricetin is an extract of grape vine tea, and is the main active component of flavonoids in rattan tea. Myricetin is a special kind of flavonoids, which can relieve alcohol poisoning, prevent alcoholic liver, fatty liver, inhibit the incidence of hepatocellular carcinoma, anti-hypertension, inhibit platelet aggregation in vitro and thrombosis in vivo, reduce blood lipids and blood sugar. It has special effects in improving the activity of SOD and protecting the liver.
1. Antibacterial effect: Pharmacological experiments show that dihydromyricetin has antibacterial effects on Bacillus subtilis, Staphylococcus aureus, Salmonella, Escherichia coli, Aerobacter, Saccharomyces cerevisiae, Rhodotorula mucus, Penicillium, Aspergillus niger, Aspergillus flavus , Mucor and Rhizopus all have bacteriostatic effects, especially on Gram-positive, Gram-negative cocci or bacilli.
2. Regulating effect on blood sugar and blood lipids: Pharmacological experiments in mice by gavage show that dihydromyricetin can significantly inhibit the increase of blood sugar in mice induced by alloxan, epinephrine, streptozotocin, etc., and at the same time increase serum insulin At the same time, the infiltration of lymphocytes in pancreatic tissue was significantly reduced, the inflammatory response was significantly reduced, and the number of pancreatic islets was significantly increased. Dihydromyricetin can reduce the content of serum triacylglycerol (TG) in streptozotocin-induced hyperglycemia rats, but has no significant effect on the content of serum total cholesterol (TC) and high density lipoprotein cholesterol (HDL2C).
3. Hepatoprotective effect: Dihydromyricetin has significant protective effect on rat hepatocytes from carbon tetrachloride poisoning injury, D2 galactosamine and lipopolysaccharide-induced liver injury in mice. Dihydromyricetin can protect the liver, accelerate the rapid decomposition of acetaldehyde, a metabolite of ethanol, and turn it into a non-toxic substance, reducing its damage to liver cells. In addition, dihydromyricetin can improve the increase of serum lactate dehydrogenase activity caused by liver cell injury, inhibit the formation of collagen fibers in hepatic M cells, so as to protect the liver and protect the liver, and greatly reduce the damage of ethanol to the liver. The normal state of the liver can be quickly restored. Microherb test proves that dihydromyricetin can protect the liver, accelerate the rapid decomposition of acetaldehyde, a metabolite of ethanol, into non-toxic substances, and reduce damage to liver cells. Dihydromyricetin works quickly and has a long-lasting effect.
4. Antioxidant effect: Dihydromyricetin with a purity of 98% can significantly inhibit the formation of malondialdehyde (MDA) in rat myocardial, liver and brain tissue homogenates, and inhibit MDA with the increase of dihydromyricetin concentration The effect of generating, 99% dihydromyricetin on the scavenging rate of diphenyltrinitrophenylhydrazine (DPPH) free radicals in the test system was increased. Dihydromyricetin can significantly inhibit the formation of MDA in oil, and the antioxidant effect is enhanced with the increase of dihydromyricetin purity (60% to 90%); it has strong antioxidant effect on animal and vegetable oils.
